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Azrolid (Azithromycin)

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Azrolid is used for treating mild to moderate infections caused by certain bacteria. It may also be used alone or with other medicines to treat or prevent certain infections in persons with advanced HIV infection. Azrolid is a macrolide antibiotic. It slows the growth of, or sometimes kills, sensitive bacteria by reducing the production of important proteins needed by the bacteria to survive.

Other names for this medication:
Altezym, Amsati, Azatril, Azidraw, Azilide, Azimax, Azimed, Azinix, Azitrim, Azitrin, Azitro, Azitropharma, Azitrotek, Azitrovid, Azromax, Aztrin, Azyter, Cinalid, Doromax, Goldamycin, Goxil, Inedol, Macrozit, Naxocina, Neofarmiz, Novatrex, Novozithron, Penalox, Rozith, Sitrox, Triamid, Vinzam, Zentavion, Zibac, Zirocin, Zithrocin, Zithrogen, Zitrim, Zomax, Zymycin

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Also known as: Azithromycin


Generic Azrolid acts as an anti-infection remedy. Generic Azrolid operates by killing bacteria which spreads by infection.

Generic Azrolid and other antibiotics don't treat viral infections (flu, cold and other).

Generic Azrolid can be successfully taken by children: who are over 1 year old in treatment of community acquired pneumonia, tonsillitis or pharyngitis, otitis media, who have allergy to penicillin.

Generic Azrolid is a macrolide antibiotic.


Follow all directions on your prescription label and read all medication guides or instruction sheets. Use the medicine exactly as directed.

Azrolid IV oral is taken by mouth. This medicine injection is given as an infusion into a vein, usually for 2 days before you switch to this medicine oral. A healthcare provider will give you this injection.

You may take Azrolid IV oral with or without food.

Shake the oral suspension (liquid) before you measure a dose. Use the dosing syringe provided, or use a medicine dose-measuring device (not a kitchen spoon).

Use Azrolid IV for the full prescribed length of time, even if your symptoms quickly improve. Skipping doses can increase your risk of infection that is resistant to medication. Azrolid IV will not treat a viral infection such as the flu or a common cold.

Store at room temperature away from moisture and heat. Throw away any unused liquid medicine after 10 days.

Detailed Azrolid dosage information


Adverse reactions experienced at higher than recommended doses were similar to those seen at normal doses particularly nausea, diarrhea, and vomiting. In the event of overdosage, general symptomatic and supportive measures are indicated as required.


Keep out of the reach of children.

Do not keep outdated medicine or medicine no longer needed.

Ask your healthcare professional how you should dispose of any medicine you do not use.

Store the medicine in a closed container at room temperature, away from heat, moisture, and direct light. Keep from freezing.

Do not refrigerate or freeze the Zmax® extended-release oral suspension. After water has been added to the powder, use the dose within 12 hours and throw away any unused liquid after your dose.

You may store the Azrolid® oral liquid at room temperature or in the refrigerator. Do not freeze the bottle. Do not keep the oral liquid for more than 10 days. Throw away any unused liquid after all doses are completed.

Detailed Azrolid dosage information

Side effects

The most common side effects associated with Azrolid are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Tell your doctor if you have ever had any unusual or allergic reaction to this medicine or any other medicines. Also tell your health care professional if you have any other types of allergies, such as to foods, dyes, preservatives, or animals. For non-prescription products, read the label or package ingredients carefully.

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Effect of streptozotocin on erythrocyte and retinal superoxide dismutase. Friction of fatty acids in nanometer-sized contacts of different adhesive strength. Effect of treatment on fertility and sexual function in males with metastatic nonseminomatous germ cell tumors of testis. A randomized, crossover study to evaluate the pharmacokinetics of amantadine and oseltamivir administered alone and in combination. Novel beta-lactamase-random peptide fusion libraries for phage display selection of cancer cell-targeting agents suitable for enzyme prodrug therapy. KC release was not prevented by the pan-caspase inhibitor, zVAD.fmk.

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A new biliodigestive anastomosis technique after hepatic artery thrombosis as what class of antibiotic is levaquin bridge to liver retransplantation: a case presentation. Bi(OTf)3 and SiO2-Bi(OTf)3 are found to effectively catalyze the Ferrier rearrangement of tri-O-acetyl glycals with different alcohols providing an effective route to 2,3-unsaturated O-glycosides with good anomeric selectivity and good to excellent yields after short reaction times. NDRG2 expression in astrocytes may play important pathological roles in cell apoptosis after stroke. Meningeal carcinomatosis as the first manifestation of malignant carcinomatosis. A limitation has been identified: all MDE tools support UML (Unified Modeling Language), which is a standard model language, but most do not support the HL7 proprietary model language. Radial Distribution Function descriptors for predicting affinity for vitamin D receptor. Phalloidin suppresses the force in nebulin-rich lamprey cardiac muscle. The Phasic triplet Markov chain, proposed in this paper, overcomes this difficulty by means of an auxiliary underlying process in accordance with the triplet Markov chains theory.

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In concordance, p73alpha decreases the p300-mediated acetylation of NF-YC, p300 competes with p73alpha for binding NF-YB, and P/CAF competes with p73alpha for binding NF-YB and NF-YC. Recurring hemolytic anemia, babesiasis, and influenza A viruses in a yak at low altitude in Nepal. The incidences of major structural defects and minor anomalies were evaluated then sonographic signs with the highest incidences were established in each of the major chromosomal defects. Importantly, we found that both CINC-1 and CINC-3 could signal to rat cortical neurons. Cysteine, known to be involved in a myriad of immuno-modulatory, anti-oxidant, and anti-carcinogenic pathways, has not been investigated in the aetiology of oesophageal or gastric cancers. Congenital abdominal wall defects are common anomalies which include gastroschisis, omphalocele and umbilical cord hernia. These objectives include the exploration of presentation of quality of life information and prospective anchor-based approaches. sex, age, and history of tuberculosis) as well as the drug resistance patterns and sources of the clinical isolates were collected. In addition, to assay any potential effects from growth in spaceflight hardware, we performed another control by growing seedlings in Petri dishes in standard laboratory conditions (termed the hardware control, HC).

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Knowledge and practice of traditional sumycin suspension drug category healers in oral health in the Bui Division, Cameroon. Distribution and origin of substance P- and neuropeptide Y-immunoreactive nerves in the guinea-pig heart. To determine the activity of p73 functional domains, we have generated stable inducible cell lines that express p73beta and various mutants deficient in one or more functional domains. CT-guided tube pericardiostomy is a safe and effective alternative to surgical drainage in the care of patients with clinically significant pericardial effusion after cardiothoracic surgery and has the additional benefit of substantial cost savings. We welcome all forms of discussion about this case and similar cases around the globe. In pediatric cases, the disease advanced to stage V within 10 years from the onset in the majority of patients. Assay development and case history of a 32K-biased library high-content MK2-EGFP translocation screen to identify p38 mitogen-activated protein kinase inhibitors on the ArrayScan 3.1 imaging platform. We estimated the comparative efficacy of antiviral drugs for prevention of vertical transmission of HBV, through a network meta-analysis of clinical trials. Interestingly, heterozygous mutation of the NKX2.1 gene in humans has been described as causing an unusual disorder from the second year of life onwards, which is mainly characterized by disturbances of motor abilities and delayed speech development.

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