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Atensia (Atenolol)
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Atensia

Atensia is used with or without other medications to treat high blood pressure (hypertension). Lowering high blood pressure helps prevent strokes, heart attacks, and kidney problems.

Other names for this medication:
Altol, Amcard, Amdip, Amdonex, Amloat, Amloheart, Amlokos, Amlong, Amlonorm, Amlop, Amlostar, Amlotin, Amodep, Ampin, Amrap, Anten, Atenex, Atepose, Atloma, Betacard, Betanif, Betanol, Biduten, Biocard, Emadine, Espil, Exlodep, Gericard, Hibesor, Hipres, Hypernorm, Hyten, Licopin, Malodip, Nipidol, Opime, Pericard, Samstar, Tecard, Teenol, Tenocard, Tensicard, Tensigard, Tinormin, Topdip, Zodipin

Similar Products:
Rocaltrol, Famvir, Sporanox, Urispas, Ticlid, Imovane, Myambutol, Uroxatral, Clomid

Also known as: Atenolol

Description

Atensia is created by pharmacy specialists to combat angina, heart attack, high blood pressure, anxiety attacks and migraine. Target of Atensia is to influence circulation. Atensia is also known as Atenolol, Hipres, Atendol, Altol, Atelol, Antipressan, Atenix, Ateno, Betacard, Blokium, Catenol, Corotenol, Notenol, Oraday. Atensia acts as an anti-angina remedy. Atensia operates by controlling and decreasing the strength of heart contractions and, thus, influencing circulation. Atensia is a beta-blocker. You can use Atensia with other anti-high blood pressure medication. Generic name of Atensia is Atenolol. Brand name of Atensia is Atensia.

Dosage

Usual Adult Dose of Atensia for Hypertension:

Initial dose: 50 mg orally once a day Maintenance dose: 50 to 100 mg orally once a day Maximum dose: 100 mg per day Comments: -If desired response not achieved after 1 to 2 weeks, increase to 100 mg may be beneficial. -Doses greater than 100 mg once a day did not result in significant additional antihypertensive effects. Use: For the treatment of hypertension alone or in combination with other antihypertensive agents.

Usual Adult Dose of Atensia for Angina Pectoris Prophylaxis:

Initial dose: 50 mg orally once a day -Increase to 100 mg orally once a day after 1 week if optimal response not achieved Maintenance dose: 50 to 200 mg orally once a day Maximum dose: 200 mg per day Comments: -Some patients may require 200 mg per day to attain optimal effect. Use: For the long-term management of angina pectoris due to coronary atherosclerosis.

Usual Adult Dose for Angina Pectoris:

Initial dose: 50 mg orally once a day -Increase to 100 mg orally once a day after 1 week if optimal response not achieved Maintenance dose: 50 to 200 mg orally once a day Maximum dose: 200 mg per day Comments: -Some patients may require 200 mg per day to attain optimal effect. Use: For the long-term management of angina pectoris due to coronary atherosclerosis.

Usual Adult Dose of Atensia for Myocardial Infarction:

50 mg orally twice a day or 100 mg orally once a day Comments: -If IV beta blockers are contraindicated or inappropriate, oral therapy should continue for at least 7 days post-myocardial infarction (MI). -Treatment with beta blockers post MI should generally continue for 1 to 3 years if there are no contraindications. Use: For the management of hemodynamically stable patients with definite or suspected acute myocardial infarction to reduce cardiovascular mortality.

Usual Geriatric Dose for Hypertension:

Initial dose: Consider reducing the starting dose to 25 mg orally once a day

Detailed Atensia dosage information

Overdose

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include extreme weakness or lack of energy, very slow heart rate, shortness of breath, or fainting.

Storage

Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Atensia are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Appropriate studies performed to date have not demonstrated geriatrics-specific problems that would limit the usefulness of atenolol injection in the elderly. However, elderly patients are more likely to have age-related liver, kidney, or heart disease, which may require caution and an adjustment of dosage in patients receiving atenolol injection .

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EPSs serve as a scaffold for cross-linking biofilm cells and support development of biofilm architecture and functions. Recently, another enzyme encoded by the mammalian immunoresponsive gene 1 (irg1), was found to decarboxylate cis-aconitate to itaconate in vitro. Chemotaxis experiments were performed using induced sputum supernatants from subjects with and without COPD as a source of chemotactic activity, and neutrophils from healthy donors as responder cells. The overall clinical experience also suggests that stem cell therapy can be safely performed, if the right cell type is used in the right clinical setting. The effects of gender, age, and body mass index on standing lumbar curvature in persons without current low back pain. Effects of introducing routinely ultrasound scanning during Ante Natal Care (ANC) clinics on number of visits of ANC and facility delivery: a cohort study.

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Impacts of Mesopredator Control on Conservation of Mesopredators and Their Prey. Differences in contractile responses to electrical stimulation and alpha-adrenergic binding sites in isolated cerebral arteries of humans, cows, dogs, and monkeys. In those patients who underwent LVR because of ischemic cardiomyopathy, the administration of milrinone achieved safe perioperative management for stable hemodynamics and reduced the postoperative doses of dobutamine and epinephrine. pylori antibodies with ABO blood group and secretor status, which implies that H. The virulence of human pathogenic fungi: notes from the South of France. To study the conformational dynamics of the empty major histocompatibility complex class I molecule comparative molecular dynamics simulations have been performed starting from the crystal structure of a peptide bound class I peptide-binding domain in the presence and absence of a peptide ligand. To identify the magnitude of changes occurring, search for potential explanations from population and clinical epidemiology and present the data available regarding etiology and treatment of PEA.

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